該分段法各段的解剖分界明確，共分為七段：C1頸段(Cervical segement)，C2巖段（Petrous segment），C3破裂（孔）段（Lacerum segment），C4海綿竇段（Cavenous segement），C5床段（Clinoidal segment），C6眼段（Opt細菌almic segement）和C7交通段（Communicating segment ）[1] 。C1頸段：頸段起于頸總動脈分叉水平，終止于頸動脈管顱外口。C2巖段：這段頸內動脈位于頸動脈管內，起于頸動脈管顱外口，終止于破裂孔后緣。C3破裂（孔）段：破裂段起于頸動脈管末端，動脈越過孔部，但不穿過這個孔，在破裂孔的垂直管內上升，向著海綿后竇，止于巖舌韌帶上緣。C4海綿竇段：此段始于巖舌韌帶上緣，止于近側硬膜環。C5床段：此段起于近側硬膜環，止于遠側硬膜環。C6眼段：該段起于遠側硬膜環，止于后交通動脈起點的緊近側。C7交通段：交通段起于緊靠后交通動脈起點的近側，止于頸內動脈分叉處。動眼神經（oculomotor nerve）為運動性神經，含有軀體運動和內臟運動兩種纖維。軀體運動纖維起于中腦動眼神經核，一般內臟運動纖維起于動眼神經副核。動眼神經自腳間窩出腦，緊貼小腦幕緣及后床突側方前行，進入海綿竇側壁上部，再經眶上裂眶，立即分為上、下兩支。上支細小，支配上直肌和上瞼提肌。下支粗大，支配下直、內直和下斜肌。由下斜肌支分出一個小支叫睫狀神經節短根，它由內臟運動纖維（副交感）組成，進入睫狀神經節交換神經元后，分布于睫狀肌和瞳孔括約肌，參與瞳孔對光反射和調節反射。動眼神經麻痹時，出現上眼瞼下垂，眼球向內、向上及向下活動受限而出現外斜視和復視，并有瞳孔散大，調節和聚合反射消失。常見的病細菌有動眼、滑車與外展神經本身炎癥而致的麻痹，急性感染性多發性神經炎，繼發于頭面部急、慢性炎癥而引起海綿竇血栓形成。

The anatomical boundaries of each segment of the segmentation method are clear and divided into seven sections: Cervical segement, Petrous segment, Lacerum segment, Cavenous segment, segement, Clinoidal segment, Optics almic segement and C7 Communicating segment [1]. C1 neck: the neck from the common carotid artery bifurcation level, terminating in the carotid artery cranial mouth. Section C2: This section of the internal carotid artery located in the carotid artery, the carotid artery in the cranial mouth, terminating in the posterior edge of the rupture. C3 Rupture (Pore): The rupture begins at the end of the carotid artery. The artery passes through the hole but does not pass through this hole. It rises in the vertical tube of the ruptured hole toward the posterior sinus of the sponge, stopping at the upper edge of the tongue. C4 cavernous sinus segment: This segment began in the upper edge of the tongue ligament, ending in the proximal dural ring. C5 bed segment: This section from the proximal dural ring, ending in the distal dural ring. C6 segment: the segment from the distal dural ring, just after the start of the posterior communicating artery close to the side. C7 traffic section: the traffic section close to the starting point of the artery immediay after the carotid artery at the bifurcation. Oculomotor nerve is a motor nerve that contains both somatic and visceral motility fibers. Somatic motor fibers start in the midbrain optic nerve nucleus, the general movement of visceral fibers from the oculomotor nerve nucleus. Occurred from the foot of the oculomotor nerve, close to the cerebellar margin and the posterior lateral approach, into the upper cavernous sinus wall, and then through the supraorbital fissure, immediay divided into upper and lower two. Upper small, dominate the rectus and levator muscle. Under the branch thick, dominated the next straight, straight and lower oblique. From the inferior oblique muscular branch of a small branch called the ciliary ganglion short root, which consists of visceral motility fibers (parasympathetic), into the ciliary ganglion neurons, distributed in the ciliary muscle and pupil sphincter, involved in pupillary pairs Light reflection and reflection adjustment. Oculomotor nerve paralysis, the upper eyelid ptosis, eyeball inward, upward and downward mobility constraints appear exotropia and diplopia, and dilated pupils, regulation and polymerization disappear. Common disease bacteria have moving eyes, block and outreach nerve inflammation itself caused by paralysis, acute infectious polyneuritis secondary to head and face acute and chronic inflammation caused by cavernous sinus thrombosis, supraorbital fissure and orbital apex Syndrome, intracranial aneurysms, intracranial tumors, other such as tuberculosis, mold, syphilis and purulent inflammation caused by skull base meningitis. Due to different bacteria, the pathogenesis is also different, such as direct compression of the tumor caused by the primary inflammation.

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